Question by johnny dean farris,: vampires are real read this :}?
 Diagnosis Porphyrin studiesPorphyria is diagnosed through spectroscopy and biochemical analysis of blood, urine, and stool. In general, urine estimation of porphobilinogen The porphyrias are a group of inherited or acquired disorders of certain enzymes in the heme bio-synthetic pathway (also called porphyrin pathway). They manifest with either neurological complications (“acute”) or skin problems (“cutaneous”), or occasionally both. The porphyrias may be classified by the primary site of the overproduction and accumulation of the porphyrins (or their chemical precursors) as hepatic porphyrias, where porphyrins are overproduced in the liver predominantly, or erythropoietic porphyrias, where the overproduction is confined to the bone marrow and the erythrocye (red blood cell) and its precursors.
The term derives from the Greek πορφύρα, porphyra, meaning “purple pigment”. The name is likely to have been a reference to the purple discolouration of feces and urine when exposed to light in patients during an attack. Although original descriptions are attributed to Hippocrates, the disease was first explained biochemically by Felix Hoppe-Seyler in 1871, and acute porphyrias were described by the Dutch physician Barend Stokvis in 1889.
The prevalence of all types of porphyria taken together has been estimated to be approximately 1 in 25,000 in the United States. The worldwide prevalence has been estimated to be somewhere between 1 in 500 to 1 in 50,000 people.
(PBG) is the first step if acute porphyria is suspected. As a result of feedback, the decreased production of heme leads to increased production of precursors, PBG being one of the first substances in the porphyrin synthesis pathway. In nearly all cases of acute porphyria syndromes, urinary PBG is markedly elevated except for the very rare ALA dehydratase deficiency or in patients with symptoms due to hereditary tyrosinemia type I. In cases of mercury- or arsenic poisoning-induced porphyria, other changes in porphyrin profiles appear, most notably elevations of uroporphyrins I & III, coproporphyrins I & III and pre-coproporphyrin.
Repeat testing during an attack and subsequent attacks may be necessary in order to detect a porphyria, as levels may be normal or near-normal between attacks. The urine screening test has been known to fail in the initial stages of a severe life threatening attack of acute intermittent porphyria.
The bulk (up to 90%) of the genetic carriers of the more common, dominantly inherited acute hepatic porphyrias (acute intermittent porphyria, hereditary coproporphyria, variegate porphyria) have been noted in DNA tests to be latent for classic symptoms and may require DNA or enzyme testing. The exception to this may be latent post-puberty genetic carriers of hereditary coproporphyria.
As most porphyrias are rare conditions, general hospital labs typically do not have the expertise, technology or staff time to perform porphyria testing. In general, testing involves sending samples of blood, stool and urine to a reference laboratory. All samples to detect porphyrins must be handled properly. Samples should be taken during an acute attack, otherwise a false negative result may occur. Samples must be protected from light and either refrigerated or preserved.
If all the porphyrin studies are negative, one has to consider pseudoporphyria. A careful medication review often will find the inciting cause of pseudoporphyria
When using the term “vampire disease”, it’s important to distinguish between the two different definitions of the term:
•Porphyric Hemophilia is a bacterial disease that humans can become infected by. An infection that is not cured will lead to vampirism.
•Sangue Debolezza is a potentially deadly, rare disease that only vampires can catch.
Porphyric Hemophilia is a condition caused by a bacterial strain carried in the blood and saliva of every vampire. A human who comes in contact with the blood or saliva of a vampire can easily become exposed to this bacteria. Once exposed, an infected human only has about 72 hours to begin antibiotic treatments before the bacteria becomes incurable.
In essence, this “vampire disease” spreads through the human’s body, changing the chemistry of the blood and skin. The first symptoms of infection may include photosensitivity or photodermatitis (sensitivity to light of the eyes o
Patients suffering from the disease were called vampires in the inquisition era because of the symptoms they displayed, many of which are now the fundamental characteristics of the fictional modern day vampire.
People suffering from the vampire disease are incredibly sensitive to the sun. So much so that once exposed to the rays of the sun, their skin would blacken, rupture, swell and eventually become disfigured.
The shrinking of the gums is another common symptom as well as the tautness of the skin, resulting in the exposure of ‘fangs’. If you are wondering if sufferers used to drink blood, then you wouldn’t be entirely wrong. Some were believed to ingest animal blood, but only because they thought it would relieve their anemia. Their urine is also purple-colored, causing many to believe that they did indeed drink blood.
Another common symptom of porphyria is being allergic to garlic. Not because they’ll be repelled and run a thousand miles at a supersonic speed, but because the c
A rare hereditary blood disease, which causes the inability of the body to reproduce heme, the component of hemoglobin, which is the major component or red blood. Lack of heme causes a porphyriac to experience blood cravings, causing the sufferer of this disease to attack people or animals for their blood in desparate attempt to replace the heme their body could not generate. A driven porphyriac would do this without knowing why he/she had done it.
Porphyria causes skin sensativity to sunlight forcing the sufferer to come out only at night. Garlic, which stimulates heme production in healthy people, contains a chemical that worses then painful symptoms of porphyria. The porphyria sufferer would avoid contact with garlic becaused it caused pain, not because he/she was a Vampire.
More severe symptoms caused by this disease are sores and scars on the skin, exessive hariness, the tightening and stretching of the gums and lips causing teeth to appear fang-like. Aslo, in more severe case
Answer by Maiingan
Uh…no I ain’t reading wall-o-text
ETA: Medical condition does NOT equal “Vampire”.
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